By J. D. Pickard, N. Akalan, V. V. Dolenc, J. J. A. Mooij
This sequence, backed via the eu organization of Neurosurgical Societies, has already develop into a vintage. regularly, one quantity is released according to 12 months. The Advances part provides fields of neurosurgery and comparable components during which vital fresh development has been made. The Technical criteria part positive aspects specified descriptions of ordinary methods to help younger neurosurgeons of their post-graduate education. The contributions are written via skilled clinicians and are reviewed through all individuals of the Editorial Board.
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Recurrent Depolarisations Following Experimental Traumatic Brain Injury (TBI) The term ‘‘peri-infarct depolarisation’’ is not strictly applicable to a depolarisation occurring spontaneously in the periphery of a traumatic contusion or intracortical haematoma, but several reports (below) of depolarisations in experimental TBI raise two questions. First, do such depolarisations have the characteristics of CSD or of PID, and second, is there evidence for similar events in the injured human brain? Until it becomes clear whether or not depolarisations around a contusion have the characteristics of an (ischaemic) PID, it seems wiser not to assign the term ‘‘PID’’ or ‘‘CSD’’ to them.
See also Fig. 5). On suprasylvian gyrus, the majority of ﬂuorescence increases are sustained, probably indicating terminal depolarisation. On the middle and posterior MG, still within MCA territory but better collateralised, ﬂuorescence increases are smaller than on SG, and not sustained. In the anterior MG, within anterior cerebral territory, ﬂuorescence transients are all decreases, indicating either oxidation of the NAD/H couple, or an increase in total haemoglobin content in the parenchymal circulation, implying vasodilation.
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Advances and Technical Standards in Neurosurgery by J. D. Pickard, N. Akalan, V. V. Dolenc, J. J. A. Mooij